How It Works
A deficiency in melatonin levels can contribute to prolonged sleep latency, inability to stay asleep, disruption to circadian rhythms and jet lag. Disordered circadian rhythm is hypothesized to contribute to increased appetite and weight gain. Melatonin also has the capability to reduce ACTH-stimulated cortisol production that has been found to interrupt sleep.
The delayed-release melatonin in SomniTR provides sustained levels for up to eight hours.
SomniTR also provides support for GABA-A receptor sites.GABA-A agonists, such as Lactium have been shown to improve the quality of sleep. ForsLean supports cAMP while pantothenic acid supports acetyl coenzyme A, both of which are important in melatonin synthesis.
SomniTR provides support for patients with sleeping problems.* It may be taken in conjunction with Lentra™, Prolent™, and/or Tranquilent™.
Explanation of Ingredients
SomniTR contains ingredients that support melatonin production by supplementing multiple steps in the serotonin-to-melatonin pathway or influence inhibitory GABAergic activity. The tablet offers both immediate-release and delayed-release melaton into keep melatonin levels steady throughout the sleep cycle.
Lactium® Acts as a GABA-A receptor agonist. Lactium has been shown to decrease stress-related symptoms in women and significantly improve sleep quality.
ForsLean® (standardized to 20% forskolin) a source of forskolin that increases cAMP. Serotonin N-acetyltransferase requires cAMP to metabolize serotonin to N-acetylserotonin, the immediate precursor to melatonin.
Pantethine Precursor to acetyl coenzyme A. Production of N-acetylserotonin requires acetyl coenzyme A.
Calcium (as Dicalcium Phosphate) potentiates the effects of cAMP.* Serotonin N-acetyltransferase requires cAMP to convert serotonin to the precursor to melatonin.
Melatonin is a neurohormone involved with circadian rhythm regulation and sleep induction.